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A case of hepatomegaly

¹ Department of Gastroenterology, William Harvey Hospital, East Kent Hospitals, Ashford, Kent, UK
² Digestive Disease Department, Brighton & Sussex University Hospitals, Brighton, East Sussex, UK
³ Department of Haematology, William Harvey Hospital, East Kent Hospitals, Ashford, Kent, UK
⁴ Department of Histopathology, Institute of Liver Studies, Kings College Hospital, London, UK

Correspondence to:
Dr D Joshi, Department of Gastroenterology, William Harvey Hospital, East Kent Hospitals, Ashford, Kent, UK; djosh78@hotmail.com

Submitted 18 June 2007
Accepted 18 July 2007

Keywords: AL amyloid; Congo red; hepatomegaly

The first 150 words of the full text of this article appear below.

A female publican in her 60s presented to clinic with abnormal liver function tests: bilirubin 17 µmol/l (normal range (NR) 0–17 µmol/l), alanine transaminase (ALT) 73 IU/l (NR 0–70 IU/l), alkaline phosphatase (ALP) 463 IU/l (NR 42–128 IU/l), glutamyl transferase (GGT) 784 IU/l (NR 2–35 IU/l), albumin 35 g/l (NR 35–50 g/l), prothrombin time (PT) 11.0 s (NR 10–12 s). She was asymptomatic with no history of weight loss or recent travel. She consumed an estimated 20 units of alcohol per week and was not taking any regular medications. Past medical history was unremarkable. The patient’s body mass index was 19 kg/m². On examination, a 10 cm non–tender liver was palpable. No stigmata of chronic liver disease were present. The full blood count, and urea and electrolytes were normal. Viral hepatitis serology was negative. Autoimmune antibody serology, serum copper, caeruloplasmin and ferritin were normal.

An ultrasound . . . [Full text of this article]